Living Well and How does the memory store in the brain?

Although I slept 1.00AM last night and got up around 9.00AM, I still feel sleepy all the time this morning. Nowadays, I always get stress about my final exams and this make me anxious which can express like cannot sleep easily and dream about many stories during sleep. To clear food stuffs in the freezer of P Sing, I found chicken that was much enough to cook one main course, so, finally, I decided to cook chicken green curry which is very tasty and fabulous in my opinion. This menu could possibly supply me for at least 3 days and I have to complete it by myself because I am living alone right now without P Sing who is in Thailand now.
As I am in the revision period, reading book is my main interest alternating with serfing the net like neuroscience websites and Pantip to answer some practical questions posted by Thai people there like, for example, how to prepare for overseas interview organized by UCL course director. Because I have experienced in this case as I was interviewed by Cardiff University, University of California, Los Angeles and Imperial College London last year, so I suggested he to organize all necessary short notes in front of him during the interview and turn the telephone speaker on in order to allow his colleagues to help suggest answers in case he cannot understand what is being asked.
Regarding my topics being read by myself now, I feel very difficult to understand the whole story of learning, memory, long term potentiation as well as long term depression which are compulsary topics and I think it will be in some parts of the final exam questions. Basically, human lern to memorize things from experience. Learning is defined by acquisition new information. And memory is the process to maintain our knowledge. Generally, memory can be classified into two stages: short-term memory and long-term memory. To transfer the information memorized in the short -term period to long-term one, we have a process called memory consolidation.

All things I mentioned above are vivid in behavioural studies. However, how can we explain clearly in terms of cellular and molecular mechanisims? Let’s me explain briefly. During the learning period, there are a huge number of synaptic transmission in our brain described by a pre-synaptic neuron releases neurotransmitter molecules from its axon terminal and such the released transmitter diffuse across synaptic cleft to bind to their target called neurotransmitter receptors which are located on surface of neuronal cell membrane of a post-synaptic neuron.
NMDA receptors and AMPA receptors are the most important glutamate receptors involving learning and memory mechanism of the neuron. As far as glutamate bind to the AMPA receptor, Na+ ions can enter into nerve cell and then this cause membrane potential become more positive voltage enough to activate the NMDA receptor. Naturally, the NMDA receptor is cloged by an Mg2+ ion in the ion channel that means the channel is not allowed to cary any ions into the neuron. Once the NMDA receptor binded by glutamate is being activated by the action potential caused by AMPA receptor, the Mg2+ ion is gone and the large amount of Ca2+ and Na+ now enter to the post-synaptic neuron.
A high level of Ca2+ in form of Calcium-Calmodulin then activate two proteins: Calcium-Calmodulin -dependent protein kinase II known as CaMKII and protein kinase C.
Now I have to continue reading and I will write about the mechanism again tomorrow.